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It should be noted that neither the pbgccs - ko1 (GSH levels of 1.0 nmoles/10 9 parasites [14]) nor the pbgr-ko mutants (GSH levels of 5.0 nmoles/10 9 parasites [15]) show a statistically significant increase in nDNA damage compared to WT (GSH levels of 7.4 nmoles/10 9 parasites [14]), demonstrating that only a drastic reduction in GSH levels (i.e
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