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In addition, A53T and A30P α-syn exhibit a stronger binding affinity for the lysosomal receptor LAMP2A compared with wild-type (WT) α-syn, so these mutant forms of α-syn are not efficiently degraded by protein clearance mechanisms, which results in increased α-syn burden of chaperone-mediated autophagy and inhibits the loading and clearance of other cargo (Cuervo et al., 2011, 2016)