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compounded under Section 503A for individual prescriptions Hepatic impairment data / no controlled pharmacokinetic studies in liver disease populations Copper clearance / 85-95% of circulating copper is bound to ceruloplasmin, which is synthesized exclusively in the liver [1] Wilson disease parallel / hepatic copper accumulation can reach 250+ mcg/g dry weight in impaired biliary excretion [2] Child-Pugh dosing / no GHK-Cu dose has been validated for class A, B, or C Monitoring intervals / no GHK-Cu-specific schedule is validated Human systemic dose / not established in peer-reviewed trials Copper testing / selected for the underlying diagnosis and interpreted in clinical context What Is GHK-Cu and How Does It Work
