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We further demonstrate that loss of TBC1D9B impairs starvation-induced autophagic flux and lysosomal proteolysis via a mechanism that depends on its GAP activity and on the presence of ARL8B

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Health status considerations: Generally healthy adults with physician clearance, individuals without contraindications (pregnancy, active malignancy, severe organ disease), those willing to commit to proper administration protocols and monitoring, and patients who understand the research-purposes-only status and informed consent requirements