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doi: 10.1007/s10549-011-1624-x 214 MehnertJMSilkAWLeeJHDudekLJeongBSLiJet al

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The most well-established mechanisms include through (1) NF-κB inhibition, (2) suppression of STING signaling, (3) interference of pro-inflammatory cytokine transcription, and (4) through NRF2 target gene signaling pathways