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To maintain a balance between its functional roles and potential for generation of harmful reactive oxygen species, cellular levels of copper are tightly regulated by a network of proteins responsible for its influx, efflux, transport and storage 1,2

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Dimethylfumarate inhibits microglial and astrocytic inflammation by suppressing the synthesis of nitric oxide, IL-1β, TNF-α and IL-6 in an in-vitro model of brain inflammation