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Hypoxia-inducible factor (HIF), the effective substrate of HIF prolyl hydroxylase (HIF-PHD), similarly activates several genes involved in glucose metabolism, intracellular acidity regulation, vasculature, iron overload, mitosis, and other physiological processes (Wang and Semenza, 1995)

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N-acetylcysteine in patients with COPD exacerbations associated with increased sputum