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Therefore, the synthesis of the primary low-molecular cellular antioxidant glutathione is interdependently linked to the folate pathway, thus any aberrant disruptions in folate metabolism can result in potentially deleterious effects as a result of an imbalance in the cellular redox state
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The body responds to oxidative stress through the action of antioxidant enzymes, such as superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GSH-Px), and non-enzymatic antioxidants [9]
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